Polycystic ovary syndrome is a common gynecological inflammation, which causes great harm to women. Today I will share with you the causes of polycystic ovary for your reference only!
I. genetic factors. It is generally believed that it is related to multiple gene abnormalities, and a few patients have chromosome abnormalities.
II. Abnormal insulin. Especially obese patients can show hyperinsulinemia and insulin resistance.
III. abnormal hormone regulation function. It may be affected by mental stress, drugs and some diseases, forming a vicious circle of excessive androgen and continuous anovulation.
IV. abnormal adrenal function. Adrenocortical hyperfunction and excessive androgen secretion can lead to anovulation and polycystic ovary like symptoms.
1. Abnormal menstruation: rare menstruation or amenorrhea (accounting for 1 / 3 of patients with amenorrhea and 90% of patients with rare menstruation). Some patients will show continuous menstruation.
2. Infertility: the most prominent feature of polycystic ovary syndrome is anovulation. Because patients with polycystic ovary syndrome cannot ovulate, they cannot conceive naturally. Patients with polycystic ovary syndrome are the most common infertility patients.
3. Influence on appearance: hyperandrogenemia is another important feature of polycystic ovary syndrome. The ovaries of patients with polycystic ovary syndrome secrete a large amount of androgen, which makes them show excessive androgen, including long beard, too much pubic hair, too much acne and so on. These situations may have a negative impact on women's appearance.
4, cause diabetes and cardiovascular and cerebrovascular diseases: in recent years, many patients with polycystic ovary syndrome have hyperinsulinemia. Hyperinsulinemia patients are prone to diabetes and cardiovascular and cerebrovascular diseases, so polycystic ovary syndrome is also a high risk factor for diabetes and cardiovascular and cerebrovascular diseases.
5, the incidence of pregnancy complications can be high: once pregnancy, the risk of pregnancy induced hypertension and gestational diabetes increases significantly.
6. Cause malignant tumor: due to the long-term and continuous stimulation of estrogen on the endometrium, it is easy to lead to endometrial hyperplasia, polyp menstruation, etc., and postmenopausal delay is easy to lead to endometrial cancer.
1. The general treatment of PCOS patients is mainly diet regulation and weight control. The decline of body mass index (BMI) will improve the reproductive outcome, which shows that weight loss can affect reproductive endocrine. It is found that weight loss can reduce insulin and free testosterone and increase SHBG. Studies on the relationship between hyperinsulinemia (fasting and postprandial) and anovulation also show that the decrease of insulin level is the reason affecting the recovery of ovulation. Ovulation induction outcomes are associated with BMI, suggesting that weight can affect pregnancy outcomes. Weight control has an impact on fertility and serious metabolic disorders.
2. As a method of weight loss, exercise uses glucose in peripheral tissues to reduce the concentration of insulin. In addition, low calorie food intake will also reduce insulin secretion. Reducing body weight can reduce the concentration of insulin in blood and increase the concentration of sex hormone binding globulin and insulin-like growth factor binding protein. As a result, ovarian androgen secretion is reduced and free testosterone in blood is reduced. A weight loss of 5% can reduce the symptoms of hyperandrogen. Therefore, the diet of patients with PCOS should be low carbohydrate and low fat. Reducing body weight through diet regulation and exercise is the basic method to improve the hyperandrogen of PCOS.
3. Engaging in daily work or regular exercise or participating in weight loss training may be beneficial to long-term health, ovulation and pregnancy. Good eating habits and exercise can promote weight loss, increase pregnancy rate and reduce treatment cost. It is a simple method to treat low fertility.
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